The pathway intermediate 2‐keto‐3‐deoxy‐L‐galactonate mediates the induction of genes involved in D‐galacturonic acid utilization in Aspergillus niger

نویسندگان

  • Ebru Alazi
  • Claire Khosravi
  • Tim G. Homan
  • Saskia du Pré
  • Mark Arentshorst
  • Marcos Di Falco
  • Thi T. M. Pham
  • Mao Peng
  • Maria Victoria Aguilar‐Pontes
  • Jaap Visser
  • Adrian Tsang
  • Ronald P. de Vries
  • Arthur F. J. Ram
چکیده

In Aspergillus niger, the enzymes encoded by gaaA, gaaB, and gaaC catabolize d-galacturonic acid (GA) consecutively into l-galactonate, 2-keto-3-deoxy-l-galactonate, pyruvate, and l-glyceraldehyde, while GaaD converts l-glyceraldehyde to glycerol. Deletion of gaaB or gaaC results in severely impaired growth on GA and accumulation of l-galactonate and 2-keto-3-deoxy-l-galactonate, respectively. Expression levels of GA-responsive genes are specifically elevated in the ∆gaaC mutant on GA as compared to the reference strain and other GA catabolic pathway deletion mutants. This indicates that 2-keto-3-deoxy-l-galactonate is the inducer of genes required for GA utilization.

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عنوان ژورنال:

دوره 591  شماره 

صفحات  -

تاریخ انتشار 2017